Defective branched chain amino acid catabolism contributes to cardiac dysfunction and remodeling following myocardial infarction.
American journal of physiology. Heart and circulatory physiology2016Wang W, Zhang F, Xia Y, et al.
Study designOther primary literature
SubjectAnimal
Abstract
cc by 以外（cc by-nc / cc by-nc-nd / NONE 等）は抄録を再掲しない。DOI/PMID へのリンクのみ表示する。
MeSH
3-Methyl-2-Oxobutanoate Dehydrogenase (Lipoamide)Amino Acids, Branched-ChainAnimalsBlotting, WesternCoronary VesselsEchocardiographyFatty AcidsGlucoseHeart FailureIn Situ Nick-End LabelingLigationMaleMiceMyocardial InfarctionMyocardiumNatriuretic Peptide, BrainProtein KinasesProtein Phosphatase 2CReal-Time Polymerase Chain ReactionReverse Transcriptase Polymerase Chain ReactionSignal TransductionTOR Serine-Threonine KinasesVentricular Dysfunction, LeftVentricular Remodeling