Chronic intermittent hypoxia induces NMDA receptor-dependent plasticity and suppresses nitric oxide signaling in the mouse hypothalamic paraventricular nucleus.
The Journal of neuroscience : the official journal of the Society for Neuroscience2010Coleman CG, Wang G, Park L, et al.
Study designOther primary literature
SubjectAnimal
Abstract
cc by 以外（cc by-nc / cc by-nc-nd / NONE 等）は抄録を再掲しない。DOI/PMID へのリンクのみ表示する。
MeSH
Analysis of VarianceAnimalsArginineBlood Gas AnalysisBlood PressureCyclic N-OxidesDizocilpine MaleateDose-Response Relationship, DrugExcitatory Amino Acid AgonistsExcitatory Amino Acid AntagonistsFree Radical ScavengersHydrogen-Ion ConcentrationHypoxiaImidazolesIn Vitro TechniquesMaleMembrane PotentialsMiceMice, Inbred C57BLMicroscopy, Electron, TransmissionN-MethylaspartateNeuronal PlasticityNeuronsNitric OxideNitric Oxide Synthase Type IParaventricular Hypothalamic NucleusReceptors, N-Methyl-D-AspartateS-Nitroso-N-AcetylpenicillamineSignal TransductionTime FactorsVasopressins