Inhibiting c-Jun N-terminal kinase partially attenuates caffeine-dependent cell death without alleviating the caffeine-induced reduction in mitochondrial respiration in C2C12 skeletal myotubes.

Biochemical and biophysical research communications2016Downs RM, Hughes MA, Kinsey ST, et al.

Study designOther primary literature

SubjectAnimal

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Study design: Other primary literature
Subject: Animal
Publication year: 2016
Source: doi.org
Abstract display: Not reproduced here
Publication status: Active
Status checked: 17 Aug 2026
Collected: 3 Aug 2026
Freshness: Current
Review stage: Automated
Record status: Published

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Abstract

cc by 以外（cc by-nc / cc by-nc-nd / NONE 等）は抄録を再掲しない。DOI/PMID へのリンクのみ表示する。

MeSH

AnimalsAnthracenesCaffeineCaspase 3Cell DeathCell LineDantroleneDose-Response Relationship, DrugEndoplasmic Reticulum StressJNK Mitogen-Activated Protein KinasesMiceMitochondriaMuscle Fibers, SkeletalProtein Kinase Inhibitors

DOI 10.1016/j.bbrc.2016.10.008

PMID 27717822

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