Branched-chain amino acid catabolic defect promotes α-cell proliferation via activating mTOR signaling.
Study designOther primary literature
SubjectAnimal
Record checks
- Study design
- Other primary literature
- Subject
- Animal
- Publication year
- 2024
- Source
- doi.org
- Abstract display
- Not reproduced here
- Publication status
- Active
- Status checked
- 17 Aug 2026
- Collected
- 3 Aug 2026
- Freshness
- Current
- Review stage
- Automated
- Record status
- Published
Abstract
cc by 以外（cc by-nc / cc by-nc-nd / NONE 等）は抄録を再掲しない。DOI/PMID へのリンクのみ表示する。
MeSH
DOI 10.1016/j.mce.2023.112143
PMID 38158148
View source →