Decreased H3K9ac level of KLF4 mediates podocyte developmental toxicity induced by prenatal caffeine exposure in male offspring rats.

Toxicology letters2019Zhu Y, Chen H, Zhao X, et al.

Study designOther primary literature

SubjectAnimal

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Study design: Other primary literature
Subject: Animal
Publication year: 2019
Source: doi.org
Abstract display: Not reproduced here
Publication status: Active
Status checked: 17 Aug 2026
Collected: 3 Aug 2026
Freshness: Current
Review stage: Automated
Record status: Published

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Abstract

cc by 以外（cc by-nc / cc by-nc-nd / NONE 等）は抄録を再掲しない。DOI/PMID へのリンクのみ表示する。

MeSH

AcetylationAnimalsCaffeineCells, CulturedCentral Nervous System StimulantsFemaleGene Expression Regulation, DevelopmentalGestational AgeGlucocorticoidsHistone DeacetylasesHistonesKidney DiseasesKruppel-Like Factor 4Kruppel-Like Transcription FactorsLysineMaleMaternal ExposureMembrane ProteinsPhenotypePodocytesPregnancyPrenatal Exposure Delayed EffectsPromoter Regions, GeneticRats, WistarReceptors, GlucocorticoidSignal Transduction

DOI 10.1016/j.toxlet.2019.07.011

PMID 31306741

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