Defective branched chain amino acid catabolism contributes to cardiac dysfunction and remodeling following myocardial infarction.

American journal of physiology. Heart and circulatory physiology2016Wang W, Zhang F, Xia Y, et al.

Study designOther primary literature

SubjectAnimal

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Study design: Other primary literature
Subject: Animal
Publication year: 2016
Source: doi.org
Abstract display: Not reproduced here
Publication status: Active
Status checked: 17 Aug 2026
Collected: 3 Aug 2026
Freshness: Current
Review stage: Automated
Record status: Published

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Abstract

cc by 以外（cc by-nc / cc by-nc-nd / NONE 等）は抄録を再掲しない。DOI/PMID へのリンクのみ表示する。

MeSH

3-Methyl-2-Oxobutanoate Dehydrogenase (Lipoamide)Amino Acids, Branched-ChainAnimalsBlotting, WesternCoronary VesselsEchocardiographyFatty AcidsGlucoseHeart FailureIn Situ Nick-End LabelingLigationMaleMiceMyocardial InfarctionMyocardiumNatriuretic Peptide, BrainProtein KinasesProtein Phosphatase 2CReal-Time Polymerase Chain ReactionReverse Transcriptase Polymerase Chain ReactionSignal TransductionTOR Serine-Threonine KinasesVentricular Dysfunction, LeftVentricular Remodeling

DOI 10.1152/ajpheart.00114.2016

PMID 27542406

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