Nitric oxide synthase 3 deficiency limits adverse ventricular remodeling after pressure overload in insulin resistance.

American journal of physiology. Heart and circulatory physiology2011Kurtz B, Thibault HB, Raher MJ, et al.

Study designOther primary literature

SubjectAnimal

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Study design: Other primary literature
Subject: Animal
Publication year: 2011
Source: doi.org
Abstract display: Not reproduced here
Publication status: Active
Status checked: 17 Aug 2026
Collected: 3 Aug 2026
Freshness: Current
Review stage: Automated
Record status: Published

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Abstract

cc by 以外（cc by-nc / cc by-nc-nd / NONE 等）は抄録を再掲しない。DOI/PMID へのリンクのみ表示する。

MeSH

Adenosine TriphosphateAnimalsDietary FatsDisease Models, AnimalEnergy MetabolismEnzyme InhibitorsGene Expression Regulation, EnzymologicHeart FailureHydrogen PeroxideHypertensionInsulin ResistanceMaleMiceMice, Inbred C57BLMice, KnockoutMitochondria, HeartMyocytes, CardiacNitric Oxide Synthase Type IIIOxidative StressPhosphorylationSerineTime FactorsUltrasonographyUncoupling AgentsVentricular Dysfunction, LeftVentricular Function, LeftVentricular Remodeling

DOI 10.1152/ajpheart.00744.2010

PMID 21856905

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