Metallothionein abrogates GTP cyclohydrolase I inhibition-induced cardiac contractile and morphological defects: role of mitochondrial biogenesis.

Hypertension (Dallas, Tex. : 1979)2009Ceylan-Isik AF, Guo KK, Carlson EC, et al.

Study designOther primary literature

SubjectAnimal

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Study design: Other primary literature
Subject: Animal
Publication year: 2009
Source: doi.org
Abstract display: Not reproduced here
Publication status: Active
Status checked: 17 Aug 2026
Collected: 3 Aug 2026
Freshness: Current
Review stage: Automated
Record status: Published

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Abstract

cc by 以外（cc by-nc / cc by-nc-nd / NONE 等）は抄録を再掲しない。DOI/PMID へのリンクのみ表示する。

MeSH

Analysis of VarianceAnimalsBiopterinsBlotting, WesternChromatography, High Pressure LiquidDisease Models, AnimalEchocardiographyGTP CyclohydrolaseMaleMetallothioneinMiceMice, TransgenicMitochondria, HeartMyocardial ContractionMyocytes, CardiacNitric OxideOxygen ConsumptionProbabilityRNARandom AllocationReverse Transcriptase Polymerase Chain ReactionSensitivity and SpecificitySugar Acids

DOI 10.1161/hypertensionaha.108.123422

PMID 19398661

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