S-nitrosylation of connexin43 hemichannels elicits cardiac stress-induced arrhythmias in Duchenne muscular dystrophy mice.

JCI insight2019Lillo MA, Himelman E, Shirokova N, et al.

Study designOther primary literature

SubjectHuman & animal

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Study design: Other primary literature
Subject: Human & animal
Publication year: 2019
Source: doi.org
Abstract display: Not reproduced here
Publication status: Active
Status checked: 17 Aug 2026
Collected: 4 Aug 2026
Freshness: Current
Review stage: Automated
Record status: Published

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Abstract

cc by 以外（cc by-nc / cc by-nc-nd / NONE 等）は抄録を再掲しない。DOI/PMID へのリンクのみ表示する。

MeSH

Adrenergic beta-AgonistsAnimalsArrhythmias, CardiacCell MembraneCell Membrane PermeabilityConnexin 43CysteineDisease Models, AnimalDystrophinEvoked PotentialsHumansIsoproterenolMaleMembrane PotentialsMiceMice, Inbred mdxMice, TransgenicMuscular Dystrophy, DuchenneMutationMyocytes, CardiacNitric OxideOocytesPatch-Clamp TechniquesXenopus laevis

DOI 10.1172/jci.insight.130091

PMID 31751316

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