Splenic B lymphocyte programmed cell death is prevented by nitric oxide release through mechanisms involving sustained Bcl-2 levels.

The Journal of clinical investigation1995Genaro AM, Hortelano S, Alvarez A, et al.

Study designOther primary literature

SubjectAnimal

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Study design: Other primary literature
Subject: Animal
Publication year: 1995
Source: doi.org
Abstract display: Not reproduced here
Publication status: Active
Status checked: 17 Aug 2026
Collected: 3 Aug 2026
Freshness: Current
Review stage: Automated
Record status: Published

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Abstract

cc by 以外（cc by-nc / cc by-nc-nd / NONE 等）は抄録を再掲しない。DOI/PMID へのリンクのみ表示する。

MeSH

AnimalsApoptosisB-LymphocytesCell SurvivalCells, CulturedCyclic GMPFlow CytometryGTP-Binding ProteinsH-2 AntigensHistocompatibility Antigens Class ILymphocyte ActivationMiceMice, Inbred BALB CMice, Inbred C57BLNitric OxideProto-Oncogene ProteinsProto-Oncogene Proteins c-bcl-2RNA, MessengerSignal TransductionSpleenTranscription, Genetic

DOI 10.1172/jci117869

PMID 7706495

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