Chronic intermittent hypoxia induces NMDA receptor-dependent plasticity and suppresses nitric oxide signaling in the mouse hypothalamic paraventricular nucleus.

The Journal of neuroscience : the official journal of the Society for Neuroscience2010Coleman CG, Wang G, Park L, et al.

Study designOther primary literature

SubjectAnimal

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Study design: Other primary literature
Subject: Animal
Publication year: 2010
Source: doi.org
Abstract display: Not reproduced here
Publication status: Active
Status checked: 17 Aug 2026
Collected: 3 Aug 2026
Freshness: Current
Review stage: Automated
Record status: Published

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Abstract

cc by 以外（cc by-nc / cc by-nc-nd / NONE 等）は抄録を再掲しない。DOI/PMID へのリンクのみ表示する。

MeSH

Analysis of VarianceAnimalsArginineBlood Gas AnalysisBlood PressureCyclic N-OxidesDizocilpine MaleateDose-Response Relationship, DrugExcitatory Amino Acid AgonistsExcitatory Amino Acid AntagonistsFree Radical ScavengersHydrogen-Ion ConcentrationHypoxiaImidazolesIn Vitro TechniquesMaleMembrane PotentialsMiceMice, Inbred C57BLMicroscopy, Electron, TransmissionN-MethylaspartateNeuronal PlasticityNeuronsNitric OxideNitric Oxide Synthase Type IParaventricular Hypothalamic NucleusReceptors, N-Methyl-D-AspartateS-Nitroso-N-AcetylpenicillamineSignal TransductionTime FactorsVasopressins

DOI 10.1523/jneurosci.3367-10.2010

PMID 20826673

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