Tumor necrosis factor-alpha reduces argininosuccinate synthase expression and nitric oxide production in aortic endothelial cells.

American journal of physiology. Heart and circulatory physiology2007Goodwin BL, Pendleton LC, Levy MM, et al.

Study designOther primary literature

SubjectAnimal

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Study design: Other primary literature
Subject: Animal
Publication year: 2007
Source: doi.org
Abstract display: Not reproduced here
Publication status: Active
Status checked: 17 Aug 2026
Collected: 13 Aug 2026
Freshness: Current
Review stage: Automated
Record status: Published

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Abstract

cc by 以外（cc by-nc / cc by-nc-nd / NONE 等）は抄録を再掲しない。DOI/PMID へのリンクのみ表示する。

MeSH

AnimalsAortaArginineArgininosuccinate SynthaseCattleCell NucleusCells, CulturedCitrullineDose-Response Relationship, DrugDown-RegulationEndothelial CellsEnzyme RepressionGenes, ReporterLuciferasesNF-kappa BNitric OxideNitric Oxide Synthase Type IIIPromoter Regions, GeneticRNA, MessengerSignal TransductionSp1 Transcription FactorSp3 Transcription FactorTranscription, GeneticTransfectionTumor Necrosis Factor-alpha

DOI 10.1152/ajpheart.01100.2006

PMID 17496212

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