Autophagy activated by GR/miR-421-3p/mTOR pathway as a compensatory mechanism participates in chondrodysplasia induced by prenatal caffeine exposure in male fetal rats.

Toxicology letters2024Han H, Shi H, Jiang L, et al.

Study designOther primary literature

SubjectAnimal

Record checks

Study design: Other primary literature
Subject: Animal
Publication year: 2024
Source: doi.org
Abstract display: Not reproduced here
Publication status: Active
Status checked: 17 Aug 2026
Collected: 3 Aug 2026
Freshness: Current
Review stage: Automated
Record status: Published

How to read this record →

Abstract

cc by 以外（cc by-nc / cc by-nc-nd / NONE 等）は抄録を再掲しない。DOI/PMID へのリンクのみ表示する。

MeSH

AnimalsAutophagyCaffeineChondrocytesFemaleMaleMicroRNAsPregnancyPrenatal Exposure Delayed EffectsRatsRats, Sprague-DawleySignal TransductionTOR Serine-Threonine Kinases

DOI 10.1016/j.toxlet.2024.05.010

PMID 38759937

View source →